Sure, Soy is the source of artificial transfats and it is often riddled with pesticides and spoiled by GMO processing. But that does not mean that in its natural, organic, fermented state it is bad for you. To this day, the researcher who dubbed its micronutrients “phyto-estrogens” regrets doing so. It led to serious and unfortunate misunderstandings. Some believe Soy causes breast cancer and messes up thyroid function. None of that is true.
Look up Adaptogen and Equol.
Adaptogen means that a micronutrient’s effects depend on how our liver modulates it. So, an unhealthy liver, which 1/3 of people have due to sugars and environmental toxins (Look up Fatty Liver), may trigger unwelcome reactions to phyto-estrogens.
Our liver takes its marching orders from the Microbiome, which also modulates phyto-estrogens. If you have healthy gut bacteria, Soy turns into Equol, a healthy Postbiotic. If your microbiome is not healthy, you may lack the specific bacteria that transforms Soy into Equol.
Stop blaming natural estrogens in food and in your body for hormonal and cancer problems. You are choking on gnats and swallowing elephants. The real culprits are Xenoestrogens, or toxic chemicals in our environment, which Soy helps eliminate. There are over 800 compounds that have an estrogen-like effect on our body. Look up heavy metals, BPA, phthalates, and pesticides for starters.
If you have been indoctrinated to believe Soy is bad for you, it is unlikely you will change your opinion after reading this. I can only hope you do your own research.
References
U.S. regulators should continue to recommend soy protein as part of heart-healthy diet
“Reuters (7/16, Rapaport) reports, “U.S. regulators should continue to recommend soy protein as part of a heart-healthy diet because it helps lower cholesterol, researchers” concluded in “an analysis of dozens of studies done in the past two decades.” For the study, investigators “pooled data from 46 soy trials cited by the” Food and Drug Administration “in its proposal to revoke permission for soy products to be marketed as heart healthy.” The analysis revealed that “soy reduced total cholesterol” and “also reduced levels of low-density lipoprotein…cholesterol.” The findings were published online in the Journal of the American Heart Association.”
Treatment with Phytoestrogens Reversed Triclosan and Bisphenol A-Induced Anti-Apoptosis in Breast Cancer Cells.
J. Europe PMC 2018 DOI: 10.4062/biomolther.2017.160 (PMID:29310425)
“Triclosan (TCS) and bisphenol A (BPA) are endocrine-disrupting chemicals that interfere with the hormone or endocrine system and may cause cancer. Kaempferol (Kaem) and 3,3′-diindolylmethane (DIM) are phytoestrogens that play chemopreventive roles in the inhibition of carcinogenesis and cancer progression. In this study, the influence of TCS, BPA, Kaem, and DIM on proliferation and apoptotic abilities of VM7Luc4E2 breast cancer cells were examined. MTT assay revealed that TCS (0.1-10 μM), BPA (0.1-10 μM) and E2 (0.01-0.0001 μM) induced significant cell proliferation of VM7Luc4E2 cells, which was restored to the control (0.1% DMSO) by co-treatment with Kaem (30 μM) or DIM (15 μM). Reactive oxygen species (ROS) production assays showed that TCS and BPA inhibited ROS production of VM7Luc4E2 cells similar to E2, but that co-treatment with Kaem or DIM on VM-7Luc4E2 cells induced increased ROS production. Based on these results, the effects of TCS, BPA, Kaem, and DIM on protein expression of apoptosis and ROS production-related markers such as Bax and Bcl-xl, as well as endoplasmic reticulum (ER) stress-related markers such as eIF2α and CHOP were investigated by Western blot assay. The results revealed that TCS, and BPA induced anti-apoptosis by reducing ROS production and ER stress. However, Kaem and DIM effectively inhibited TCS and BPA-induced anti-apoptotic processes in VM7Luc4E2 cells. Overall, TCS and BPA were revealed to be distinct xenoestrogens that enhanced proliferation and anti-apoptosis, while Kaem and DIM were identified as natural chemopreventive compounds that effectively inhibited breast cancer cell proliferation and increased anti-apoptosis induced by TCS and BPA.”
Fermented Soy Product Intake Is Inversely Associated with the Development of High Blood Pressure
J. Nutr. 2017 147: 1749–1756
“A Systematic Review of the Effects of Plant Compared with Animal Protein Sources on Features of Metabolic Syndrome J. Nutr. March 1, 2017 vol. 147 no. 3 281-292 plant protein (wheat gluten, soy protein) intake as part of a mixed meal resulted in a lower postprandial insulin response than did whey. This systematic review provides some evidence that the intake of soy protein associated with isoflavones may prevent the onset of risk factors associated with cardiovascular disease, i.e., hypercholesterolemia and hypertension, in humans. However, we were not able to draw any further conclusions from the present work on the positive effects of plant proteins relating to glucose homeostasis and body composition.”
Urinary Phytoestrogen Concentrations Are Not Associated with Incident Endometriosis in Premenopausal Women
J. Nutr. 2017 147: 227–234
